1. !? ??
    @ -
    : 189
    : 18.07.2019, 19:24
  2. .
    qwert -
    : 37
    : 06.03.2018, 18:06
  3. : 1
    : 31.07.2004, 03:43
  4. : 1
    : 24.07.2004, 08:42
  5. : 1
    : 01.07.2004, 06:36
  1. #106
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  2. #107
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    http://forums./showpost.php?p=19346&postcount=30

    http://forums./showpost.php?p=19412&postcount=45

    http://forums./showpost.php?p=19417&postcount=46

    http://forums./showpost.php?p=19440&postcount=62

    http://forums./showpost.php?p=19443&postcount=63

    http://forums./showpost.php?p=19445&postcount=64



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    http://www.medac.de/medac_international/txt_files/d_indications/102863791210085.htm

    Persistence

    Not recognized or recognized but not sufficiently treated C. trachomatis infections may convert into a persistent state. At this state the intracellular life cycle is characterized by the development of atypical, metabolically inert and thus antibiotic resistant inclusions. This biologic behaviour correlates with the clinical course after acute symptomatic disease, namely persistence or recurrence of symptoms which are not treatable with antibiotics. At the persistent state C. trachomatis downregulates the major outer membrane protein (MOMP) expression, still expresses continuously lipopolysaccharide (LPS) and overexpresses heat shock protein 60 (HSP60). HSP60 is a highly immunogenic protein that has been implicated in the pathogenesis of chronic inflammatory chlamydial diseases.



    http://arthritis-research.com/content/4/1/5

    However, antibiotic treatment of persistent organisms in the synovium has proved to be disappointing;



    http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=375316

    Without additional support from the host immune system, both persistent and static bacteria remain viable and may resume normal growth in changed conditions. Unlike most bacteria, the intracellular niche of persistent chlamydiae partially protects them from the immune system and thus can lead to complete failure of therapy. We have shown in this in vitro study that subinhibitory concentrations of antibiotics can induce persistence.



    http://www.jibtherapies.com/content/2/1/4

    These results argue that there is an important host-parasite relationship between chlamydial genital strains and the human host that determines organotropism of infection and the pathophysiology of disease. It was speculate that this relationship involves the production of indole by components of the vaginal microbial flora, allowing Chlamydiae to escape IFN-gamma-mediated eradication and thus establish persistent infection.



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  3. Sunspot
    #108
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    Uvazhaemy Vladimir levich,

    Standarty lechenia boleznei vsego lish ktrazhenie luchih iz izvestnykh metodov ih lecheniya. Esli standartov net ili ih ne znayut, te zhe hlamidii nachinayt lechit kerosinom ili ozonom. Neadekvatnya terapia porozhdaet v tom chisle i rezistentnost. Kstati zaregistrirovannyh sluchaev antibitikorezistentnosti hlamidi po preznemu ne mnogo i neoslozhnennye hlamidiozy uspeshno lechatsya za 1 den bolee chem v 80% sluchaev bez vsakogo ozona/vo!benzina/vo!kerosina.



    Vashu ocherednuyu provokatsiyu razreshite rascenit kak popytku kosvennoi reklamy ozonoterapii dlya lecheniya infektsii. Poskolku effectivnos OT v lechenii lubogo zabolevahiya nikem oficialno ne priznana - vy opyat pytaetes navredit lyudam, zanimayas fakticheskim moshennichestvom. Esli rossiskomu zakonodatelstvu eto vse ravno, to mne net. Esche odna takaya provokatsia - s moderiruemogo mnoi foruma budu udalyat vashi ozonovye bredni bez preduprezhdeniy.



    Izvinyaus za latinitsu, pishu iz Germanii.

  4. 1
    #109
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    Dear Vladimir levich? What is the meaning of "usrednennye issledovaniya"? And how can we call the "trials" you are usually refer on - "ahineynie" or may be you have a special name for all those garbage you usually use as an arguments? To support immune systems? Well, no questions its a good idea! But the problem is what do you mean when you are going to "support" and/or "correct" it and what methodology are you going to employ? Ozone? Come on, Sir! Do you have reliable data showing that ozone make a real difference in treating chlamydia infection? The answer is certainly no! Please do not waste our time talking about nothing.

  5. NL
    #110
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  6. stiker
    #111
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    Ph.D. in biochemistry does not probably permit to feel the difference between suggested (proposed) and actual effect of treatment. Whatever happens with t-cells antibacterials works for chlamydia in the range up to 95%. What about ozone?



    P.S. How, Dear Vladimir levich, about my remarks concerning ozone odor in German medicine? I've checked the second huge bookstore. Nothing about ozone! Secret weapon?

  7. Tito
    #112
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  8. sergeitch
    #113
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    The problem, Vladimir levich, is that doctors in Germany have absolutely the same perception about ozone therapy like in any civilized country, no difference. You were trying to to argue that ozone in Gremany is used almost anywhere in ofiicial medicine, including emergency and University hospitals facilities. This is false! And absence on widely spread textbooks on ozone just illustrates that nobody seriously interested in this issue.



    You and Mr. Dvoryanchikov and all other quackers in the world are using the same ideology. If we follow your ideology we can state that if the person have no evidence of cancer, he/she should be given a "prophylactic tratment" for cancer anyway just to "improve the immune systems". No mater what kind of "treatments" are proposed by another medical gangster.



    I've never said that the immune system does not exist. I'm just doubting that any "immune correction/stimulation" can realy help in improving the current results of antibacterial therapy for chlamydiae.

  9. moymujnumizmat
    #114
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  10. abyss
    #115
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  11. Leshiy
    #116
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    #118
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    #119
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    : 50-70% .

  15. nikat
    #120
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